The Sun is by far the largest object in the solar system. It contains more than 99.8% of the total mass of the Solar System (Jupiter contains most of the rest).

It is often said that the Sun is an “ordinary” star. That’s true in the sense that there are many others similar to it. But there are many more smaller stars than larger ones; the Sun is in the top 10% by mass. The median size of stars in our galaxy is probably less than half the mass of the Sun. A process called fusion heats the Sun. Fusion happens in the core of the Sun. It is very hot there. In fact, the core’s average temperature is around 27,000,000 degrees Fahrenheit. The surface of the Sun is cool compared with the core, only 10,000 degrees Fahrenheit.

This fusion process not only heats the Sun, it makes the sunlight we see here on Earth. This sunlight travels the speed of light which is 186,282 miles per second or 299,792,458 meters per second. This means the light from the Sun takes 8.4 minutes to travel 93 million miles to Earth. If the world’s fastest land animal were to travel that same distance, it would take a cheetah over 151 years to reach the Earth running about 70 mph nonstop!

Ultraviolet radiation: Invisible rays that are part of the energy that comes from the sun. Ultraviolet radiation can burn the skin and cause skin cancer.

Ultraviolet radiation is made up of three types of rays — ultraviolet A, ultraviolet B, and ultraviolet C. Although ultraviolet C is the most dangerous type of ultraviolet light in terms of its potential to harm life on earth, it cannot penetrate earth’s protective ozone layer. Therefore, it poses no threat to human, animal or plant life on earth.

Ultraviolet A and ultraviolet B, on the other hand, do penetrate the ozone layer in attenuated form and reach the surface of the planet. Because ultraviolet A is weaker than ultraviolet B, scientists long blamed ultraviolet B as the sole culprit in causing skin cancer in persons with a history of sunburn and repeated overexposure to ultraviolet radiation. Recent research, however, has also implicated ultraviolet A as a possible cause of skin cancer.

Photosensitivity refers to various symptoms, diseases and conditions caused or aggravated by exposure to sunlight.

• A rash due to photosensitivity is a photodermatosis (plural photodermatoses).
• If the rash is eczematous, it is a photodermatitis.
• A chemical or drug that causes photosensitivity is a photosensitiser.
• A phototoxic reaction to a photosensitiser results in an exaggerated sunburn reaction and no immune reaction is involved.
• A photoallergic reaction to a photosensitiser results in photodermatitis and is due to delayed hypersensitivity reaction.
• A photoexacerbated condition describes a flare of an underlying skin disease on exposure to sunlight.

Photosensitivity is characterized into many groups:

• Polymorphic light eruption (PLE):

PLE generally affects adult females aged 20–40, although it sometimes affects children and males (25%). It is particularly common in places where sun exposure is uncommon, such as Northern Europe, where it is said to affect 10–20% of women holidaying in the Mediterranean area. It is less common in Australasia. It has also been reported to be relatively common at higher altitudes compared to sea level.

PLE can occur in all races and skin phototypes and may be more prevalent in skin of colour than in white skin. There is a genetic tendency to PLE, and it is sometimes associated with or confused with photosensitivity due to lupus erythematosus (which generally is more persistent than PLE).

Some patients experience PLE during phototherapy, which is used to treat skin conditions such as psoriasis and dermatitis.

There are three types of UV rays:

Ultraviolet A (UVA): UVA rays penetrate deep into the layers of the skin and lead to premature signs of aging, which include fine lines and wrinkles. It is important to note that the amount of UVA stays relatively consistent throughout the year, and exceeds the amount of UVB in both summer and winter. Even on a cloudy day, 80% of the sun’s UVA rays pass through the clouds and reach our skin and eyes. It’s also important to know that tanning beds work by emitting primarily UVA rays.

Ultraviolet B (UVB): UVB rays are the primary cause of sunburn, thickening of the skin, and several types of skin cancers – including melanoma. UVB rays penetrate the outermost layer of skin and cause damage to skin cells. UVB also can cause damage to eyes and the immune system.

Ultraviolet C (UVC): UVC is the strongest of the UV spectrum radiation. Fortunately, UVC rays do not reach the earth’s surface because it is blocked by the ozone layer of the atmosphere. The only way we can be exposed to UVC radiation is from an artifical source, such as a lamp or laser. UVC rays can cause severe skin burns and eye injuries even when exposed for only a few seconds. Since the penetration depth of UVC rays is very low, these injuries usually resolve within a week with virtually no risk of long-term health risks (skin cancer, cateracts, vision loss).

Causes:

Genetic factors may be important with many affected individuals reporting a family history of PLE. Native Americans have a hereditary form of PLE (actinic prurigo).

PLE is caused by a delayed hypersensitivity reaction to a compound in the skin that is altered by exposure to ultraviolet radiation (UVR). UVR leads to impaired T cell function and altered production of cytokines in affected individuals. There is a reduction in the normal UV-induced immune suppression in the skin. This has been suggested to be either due to oestrogen or deficiency of vitamin D.

The rash is usually provoked by UVA (in 90%). This means the rash can occur when the sunlight is coming through window glass, and that standard sunscreens may not prevent it. Occasionally, UVB and/or visible light provoke PLE.

PLE may be a rare occurrence in the individual concerned or may occur every time the skin is exposed to sunlight. In most affected individuals, it occurs each spring, provoked by several hours outside on a sunny day. If further sun exposure is avoided, the rash settles in a few days and is gone without a trace within a couple of weeks. It can recur next time the sun shines on the skin. However, if the affected area is exposed to more sun before it has cleared up, the condition tends to get more severe and extensive with longer to heal.

Stay tune tomorrow for part II on Ultralight rays from the Sun to Sunburn and Types of photosensitivity for some!

“The University of Michigan recommends the following: there are very bicycle-friendly communities. It is, however, very important that bicyclists (and motorists) understand the rules of the road and how to stay safe while biking in the city. With an abundance of restaurants and shops downtown and various city parks to explore. Did you know that city ordinances require motorists to stop for pedestrians who are at or in crosswalks?  Walk with a friend, use designated sidewalks or paths, cross at designated crosswalks and don’t just look at signs but use your eyes looking both ways also.As we make the switch from warm to hot weather, it’s important to take the time to review tips for staying safe in the heat.  This means wear sunscreen for skin protection, drink plenty of water, limit alcoholic beverages, dress in loose-fitting, lightweight and light-colored clothing, and limit exercise outdoors in extreme heat.Summertime is often paired with grilling outdoors so this means place grills a minimum of 36″ away from your home, deck railings, and out from under eaves and overhanging branches, set up your grill on a flat, stable surface to prevent tipping, operate your grill safely by checking for leaks and opening the lid before lighting, maintain your grill by cleaning and inspecting it regularly and always have a fire extinguisher nearby.  Of course don’t forget the supervision of children and pets in also establishing a safe zone of at least three feet around the grill, where children and pets are not allowed.”

Division of Public Safety and Security University of Michigan –  DPSS (https://news.dpss.umich.edu/2024/06/1391)

“May sound so basic but not always followed so as a reminder to let the public know the best way to prevent HIV is to understand how the virus is transmitted and take steps to reduce your risk. If you’re living with HIV, understanding these practices can help prevent transmission to others.

HIV can’t be transmitted through saliva or skin-to-skin contact, such as hugging or shaking hands. The virus can only be transmitted by exchanging certain bodily fluids, including genital secretions and blood.

As a result, transmission most frequently occurs during condomless sex or shared use of syringes and other drug injection equipment.

Adopting certain harm reduction strategies, including safer sex and safer substance use or better yet no substance use, can help reduce the risk of contracting or transmitting the virus.

Although you have up to 72 hours to begin PEP, the medication is less likely to be effective over time. Additional medication must also be taken consistently and correctly for 28 days.

If you don’t have a primary care doctor or another healthcare professional to reach out to, you may be able to get a prescription for PEP at your local: health department, sexual health clinic, urgent care center or emergency room.

Know approximately 1.2 million people in the United States have HIV, according to HIV.gov. Of those people, 13% do not know they have it.  In 2021, there were 1,086,806 people living with HIV in the U.S. In 2021, 36,126 people were newly diagnosed with HIV.“

healthline (https://www.healthline.com/health/hiv-aids/hiv-prevention/hiv-prevention?utm_source=google&utm_medium=cpc&utm_cmpid=20958361886&utm_adgid=156321296365&utm_adid=688286705703&utm_network=g&utm_device=c&utm_keyword=&utm_adpos=&utm_gclid=EAIaIQobChMI8fKFhNj0hgMV-2BHAR2JDwgDEAMYASAAEgL8BPD_BwE&gad_source=1&gclid=EAIaIQobChMI8fKFhNj0hgMV-2BHAR2JDwgDEAMYASAAEgL8BPD_BwE#takeaway)

Acquired immunodeficiency syndrome (AIDS) is a chronic, potentially life-threatening condition caused by the human immunodeficiency virus (HIV). By damaging your immune system, HIV interferes with your body’s ability to fight infection and disease.

How HIV is transmitted:

HIV is transmitted via the exchange of body fluids—such as pre-ejaculate fluid, semen, vaginal fluid secretions, blood, or breast milk. Within these bodily fluids, HIV is present as both free virus particles and virus within infected immune cells.

The symptoms and when they develop:

Within a month or 2 of contracting HIV, about 40 to 90-percent of those afflicted suffer from flu-like symptoms including the following:

Fever, fatigue, achy muscles, swollen lymph glands, sore throat, headache, skin rash, dry cough, nausea, rapid weight loss, night sweats, frequent yeast infections (for women), cold sores, and eventually, pneumonia.

Symptoms

The symptoms of HIV and AIDS vary, depending on the phase of infection.

Primary infection (Acute HIV)

Some people infected by HIV develop a flu-like illness within two to four weeks after the virus enters the body. This illness, known as primary (acute) HIV infection, may last for a few weeks. Possible signs and symptoms include:

• Fever
• Headache
• Muscle aches and joint pain
• Rash
• Sore throat and painful mouth sores
• Swollen lymph glands, mainly on the neck
• Diarrhea
• Weight loss
• Cough
• Night sweats

These symptoms can be so mild that you might not even notice them. However, the amount of virus in your bloodstream (viral load) is quite high at this time. As a result, the infection spreads more easily during primary infection than during the next stage.

Clinical latent infection (Chronic HIV)

In this stage of infection, HIV is still present in the body and in white blood cells. However, many people may not have any symptoms or infections during this time.

This stage can last for many years if you’re not receiving antiretroviral therapy (ART). Some people develop more severe disease much sooner.

Symptomatic HIV infection

As the virus continues to multiply and destroy your immune cells — the cells in your body that help fight off germs — you may develop mild infections or chronic signs and symptoms such as:

• Fever
• Fatigue
• Swollen lymph nodes — often one of the first signs of HIV infection
• Diarrhea
• Weight loss
• Oral yeast infection (thrush)
• Shingles (herpes zoster)
• Pneumonia

Progression to AIDS

Thanks to better antiviral treatments, most people with HIV in the U.S. today don’t develop AIDS. Untreated, HIV typically turns into AIDS in about 8 to 10 years.

When AIDS occurs, your immune system has been severely damaged. You’ll be more likely to develop opportunistic infections or opportunistic cancers — diseases that wouldn’t usually cause illness in a person with a healthy immune system.

The signs and symptoms of some of these infections may include:

• Sweats
• Chills
• Recurring fever
• Chronic diarrhea
• Swollen lymph glands
• Persistent white spots or unusual lesions on your tongue or in your mouth
• Persistent, unexplained fatigue
• Weakness
• Weight loss
• Skin rashes or bumps

When to see a doctor

If you think you may have been infected with HIV or are at risk of contracting the virus, see a doctor as soon as possible.

So what is the answer to prevent this from happening by doing a few things:

-In safe sex from the front (through the vagina) or back (through the rectum-buttock); no matter what sex preference you are.

-When you use various sex partners get checked every 6 months to a year and have sex safely.

-The + HIV pt or now AIDS pt be compliant with the medications and RX the M.D. gives you if you chose not to do so previously.

Unfortunately like many who chose to do unsafe sex in getting this or worse than that those who got the disease by getting it through someone else when they didn’t have it is a shame.  Other ways of transmitting HIV/AIDS or getting it is by getting a blood transfusions (which is rare) but seen more frequently than transfusions would be sharing needle sticks, or even sex partners who didn’t tell their sex partner they had it, which it the worst someone could do to someone else. There are the those who got this disease innocently with not doing unsafe sex but for that percentage it is much lower than those who caused this disease  to spread unsafe sex or sharing needles with others who had the HIV/AIDS which caused the amount to go up higher in the USA and all over for being foolish. To all the people in the world let’s deal with this disease safely for yourselves and others around you. Let us all make a safer world for this is one small way which is greater than you may think.

HIV is a sexually transmitted infection (STI). It can also be spread by contact with infected blood or from mother to child during pregnancy, childbirth or breast-feeding. Without medication, it may take years before HIV weakens your immune system to the point that you have AIDS.

There’s no cure for HIV/AIDS, but medications can dramatically slow the progression of the disease. These drugs have reduced AIDS deaths in many developed nations. Luckily, many individuals who are diagnosed early can live a long, productive life with HIV thanks to a combination of highly active anti-retroviral drug therapy, which prevents to progression to AIDS.

“National HIV Testing Day (NHTD) encourages HIV testing as a critical tool to help end the HIV epidemic in the United States. Observed annually on June 27, the 2024 NHTD theme is Level Up Your Self-Love: Check Your Status, emphasizing self-compassion, self-respect, and self-love in honoring health needs by getting an HIV test. When someone knows their HIV status, they can choose options to stay healthy.

According to the Centers for Disease Control and Prevention (CDC), in 2022, an estimated 1.2 million people in the United States had HIV, but 13 percent of people with HIV did not know their HIV status. HIV testing is the pathway to engage people in care and help them stay healthy, regardless of the test result. People who have a positive HIV test can start antiretroviral therapy (ART) to stay healthy. People who have a negative HIV test can learn how to access HIV prevention options like pre-exposure prophylaxis (PrEP). CDC recommends that everyone aged 13–64 get tested for HIV at least once as part of routine health care. People with certain risk factors—such as having sex with someone who has HIV, sharing needles or drug injection equipment, or being treated for another sexually transmitted infection (STI)—should be tested for HIV at least once per year.”

HIVinfo.NIH.gov (https://hivinfo.nih.gov/understanding-hiv/hiv-aids-awareness-days/national-hiv-testing-day)

Where does HIV/AIDS come from for staters?  Here is the history we have learned over the years about this virus:

Well the Aids Institute states “Scientists identified a type of chimpanzee in West Africa as the source of HIV infection in humans. They believe that the chimpanzee version of the immunodeficiency virus (called simian immunodeficiency virus or SIV) most likely was transmitted to humans and mutated into HIV when humans hunted these chimpanzees for meat and came into contact with their infected blood. Over decades, the virus slowly spread across Africa and later into other parts of the world.

The earliest known case of infection with HIV-1 in a human was detected in a blood sample collected in 1959 from a man in Kinshasa, Democratic Republic of the Congo. (How he became infected is not known.) Genetic analysis of this blood sample suggested that HIV-1 may have stemmed from a single virus in the late 1940s or early 1950s.

We know that the virus has existed in the United States since at least the mid- to late 1970s. From 1979–1981 rare types of pneumonia, cancer, and other illnesses were being reported by doctors in Los Angeles and New York among a number of male patients who had sex with other men. These were conditions not usually found in people with healthy immune systems.

In 1982 public health officials began to use the term “acquired immunodeficiency syndrome,” or AIDS, to describe the occurrences of opportunistic infections, Kaposi’s sarcoma (a kind of cancer), and Pneumocystis jirovecii pneumonia in previously healthy people. Formal tracking (surveillance) of AIDS cases began that year in the United States.

What is the actual virus causing AIDS?

In 1983, scientists discovered the virus that causes AIDS. The virus was at first named HTLV-III/LAV (human T-cell lymphotropic virus-type III/lymphadenopathy-associated virus) by an international scientific committee. This name was later changed to HIV (human immunodeficiency virus).

For many years scientists theorized as to the origins of HIV and how it appeared in the human population, most believing that HIV originated in other primates. Then in 1999, an international team of researchers reported that they had discovered the origins of HIV-1, the predominant strain of HIV in the developed world. A subspecies of chimpanzees native to west equatorial Africa had been identified as the original source of the virus. The researchers believe that HIV-1 was introduced into the human population when hunters became exposed to infected blood.”

HIV is a lenti virus which is a subgroup of the retrovirus.

For starters HIV affects the white blood cells (WBCs) in this sense:

We have types of WBC’s and one type is called CD4. That is the cell the HIV binds to when it gets in your blood stream. Another name for them is T-helper cells. CD4 cells are made in the spleen, lymph nodes, and thymus gland, which are part of the lymph or infection-fighting system. CD4 cells move throughout your body, helping to identify and destroy germs such as bacteria and viruses.  Without going into specific medical terminology there is replication of this virus in the DNA and RNA killing out the normal white blood cell type of CD4 and other things like macrophages which are reproduced into CD4 cells with the virus with the body not knowing or able to detect this error in DNA and RNA of the CD4 white blood cells which ending line allows the immunity to go down. Remember the HIV virus binds to the surface of the CD4 cells. The virus entering CD4 cells now allows them to become a part of CD4 replicated cells regarding their make up due to changes made in the DNA and RNA cause the virus has invaded the bloodstream. As CD4 cells multiply to fight infection, they also make more copies of HIV now. Continuing to replicate, leading to a gradual decline of CD4 cells in decreasing that individuals immunity to infection.

The 2 types of HIV:

There are two types of HIV that have been characterized: HIV-1 and HIV-2.

HIV-1 is the virus that was initially discovered and termed both LAV and HTLV-III. It is more virulent, more infective and is the cause of the majority of HIV infections globally. The lower infectivity of HIV-2 compared to HIV-1 implies that fewer of those exposed to HIV-2 will be infected per exposure. Because of its relatively poor capacity for transmission, HIV-2 is largely confined to West Africa.

What is CD4 and how it helps the doctor:

The CD4 count measures the number of CD4 cells in a sample of your blood drawn by a needle from a vein in your arm. Along with other tests, the CD4 count helps tell how strong your immune system is. By knowing this when the blood test is done the CD4 helps the following, for the doctor:

-it indicates the stage of your HIV disease

-it guides the treatment

– it predicts how your disease may progress.

**Keeping your CD4 count high can reduce complications of HIV disease and extend your life.**

”Recent years have witnessed substantially increased research regarding sex differences in pain. The expansive body of literature in this area clearly suggests that men and women differ in their responses to pain, with increased pain sensitivity and risk for clinical pain commonly being observed among women. Also, differences in responsivity to pharmacological and non-pharmacological pain interventions have been observed; however, these effects are not always consistent and appear dependent on treatment type and characteristics of both the pain and the provider. Although the specific aetiological basis underlying these sex differences is unknown, it seems inevitable that multiple biological and psychosocial processes are contributing factors. For instance, emerging evidence suggests that genotype and endogenous opioid functioning play a causal role in these disparities, and considerable literature implicates sex hormones as factors influencing pain sensitivity. However, the specific modulatory effect of sex hormones on pain among men and women requires further exploration. Psychosocial processes such as pain coping and early-life exposure to stress may also explain sex differences in pain, in addition to stereotypical gender roles that may contribute to differences in pain expression.”

National Library of Medicine – NIH (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3690315/)