“Coronary artery disease (CAD)

• Coronary heart disease is the most common type of heart disease. It killed 371,506 people in 2022.1
• About 1 in 20 adults age 20 and older have CAD (about 5%).3
• In 2022, about 1 out of every 5 deaths from cardiovascular diseases (CVDs) was among adults younger than 65 years old.1

Heart attack

• In the United States, someone has a heart attack every 40 seconds.3
• Every year, about 805,000 people in the United States have a heart attack.3 Of these, 605,000 are a first heart attack, and 200,000 happen to be people who have already had a heart attack.3
• About 1 in 5 heart attacks are silent—the damage is done, but the person is not aware of it.”

Centers for Disease Prevention – CDC (Heart Disease Facts | Heart Disease | CDC)

This year marks the 50th Anniversary of American Heart Month. For the past 55 years, the American Heart Association (AHA) has used the month of February to partner with the media, medical providers and community organizations to spread the word about heart disease prevention and treatment. Heart disease is a leading cause of death for both men and women. Over the years, the American Heart Association has sponsored awareness and education campaigns as well as medical research funding, investing more than $3.5 billion into studies. According to the AMA, this is the most amount of funding of any entity outside the federal government.

The AHA provides the following reminders to encourage you to live a heart-healthy lifestyle:

• Watch your weight.
• Quit smoking and stay away from secondhand smoke.
• Control your cholesterol and blood pressure.
• If you drink alcohol, drink only in moderation.
• Get active – regular exercise is a verty important of heart health.
• Eat healthy.

Heart Health Facts

• Heart disease & stroke kill about 30 NC women/day.
• Nearly half of African American women live with heart disease.
• About 23% of adult men and about 18% of adult women smoke.
• Stroke is among the Top 5 Cause of Death for Women in almost every state.
• Overweight women are 18%-30% more likely to have babies with heart defects.
• 22% of schools do not require physical education.
• Nearly 10 million kids and adolescents ages 6 – 19 are considered overweight or obese.
• Each day, only 2% of children receive the right amount of fruit and veggies.

“Knowledge is power. If you understand the risks for heart attack, you can take steps to improve your health.

Risk factors are traits and lifestyle habits that can increase your chance of having a heart attack. So, it’s important to know them. You can change some risk factors, some you can’t.

It’s important to know your risk factors for a heart attack.

• You can control some risk factors, such as tobacco use, physical activity, blood pressure, blood cholesterol and blood glucose.
• Work with your health care team to manage your risk of heart disease.”

American Heart Association (Understand Your Risks to Prevent a Heart Attack | American Heart Association)

Continuation of February being the month of the heart I decided to cover the risks of a myocardial infarction (heart attack).

People who are at risk for the development of coronary artery disease and myocardial infarction include those who fall into any of the categories listed below:

-People with a history of heart disease.

-Males.

-Smokers.

-People with high cholesterol.

-People with high blood pressure.

-Obese people.

-People with diabetes.

-People who suffer stress.

-People who live a sedentary life style.

-Heredity is a powerful factor that contributes to early heart disease. Being male is a risk factor, but the incidence of heart disease in women increases dramatically after menopause.

-The risk factors to concentrate upon are those that can be modified. These include cigarette smoking, high blood pressure, cholesterol, obesity, sedentary life style and stress. Cigarette smoking causes many deaths from myocardial infarction and other heart diseases. Smoking contributes to almost half of the heart attacks of women under age 55.

-Stopping smoking can greatly reduce your chances of having a heart attack. Controlling blood pressure can reduce your risk of heart attack. Lowering cholesterol to safe levels through diet and medications can reduce your risk and may even lead to some regression of the plaques already present. Lean body weight and a regular exercise program are helpful.

-If you are diabetic, precise control of your diabetes will help reduce your risk of blood vessel damage due to diabetes. Stress is a risk factor that is common, difficult to quantify and difficult to control effectively over time. Methods of stress reduction include meditation, regular exercise, time management, and a supportive environment.

How is a heart attack diagnosed?

Chest pain is the most common symptom of a heart attack. The chest pain is usually a burning or pressure sensation beneath the mid or upper breast bone. The pain may radiate into the upper mid back, neck, jaw or arms. The pain may be severe but often is only moderate in severity.

There may be associated shortness of breath or sweating. If patients have had angina previously, the heart attack pain will feel the same as their usual angina only stronger and more prolonged. If you have a pain like this that lasts longer than 15 minutes, it is best to be evaluated immediately.

Calling your medic unit is the fastest and safest way to ask for help. If you have symptoms like this that wax and wane, this is often a warning sign that a heart attack is about to occur and prompt medical attention is needed.

Once you are in an emergency room or a doctor’s office an electrocardiogram (ECG or EKG) will be obtained. This is often helpful in diagnosing a heart attack. Sometimes, however, the test is normal even when the heart injury is present but usually a great diagnostic tool.

When heart cells die, certain enzymes present in heart cells are released into the bloodstream that serve as a marker of heart injury (troponin I and CPK or CK-MB). These enzymes can be measured by blood tests. The amount of enzyme released into the blood stream also helps assess how much heart damage has occurred.

TREATMENT:

The best way to limit the size of a heart attack is to restore the flow of blood to the heat muscle as fast as possible. There are two basic methods to do this.

Because most heart attacks are caused by clots forming within the coronary artery, dissolving the clot quickly will restore blood flow. Drugs called thrombolytics are quite effective.

The sooner these drugs are given, the quicker the blood flow will be restored. An alternative method involves the use of balloon angioplasty.

This involves taking the heart attack victim promptly to the cardiac cath lab in the hospital.

An angiogram is performed to show the blocked blood vessel leading to the heart attack. Then a balloon catheter is placed across the blockage and flow is restored.

Sometimes a stent (a device that assists in holding the blood vessel open) is placed to create a large channel.

Smaller heart attacks, often those not producing significant abnormalities on the ECG are often treated with bedrest and blood thinners such as heparin as well as drugs to reduce the work the heart does.

These heart attacks are called non-transmural myocardial infarctions. Before discharge, x-ray studies of the heart arteries are often carried out to see if angioplasty or surgery will be necessary.

Following thrombolytic (clot reducing) therapy, angiogram are often performed to outline the coronary anatomy to help determine if additional therapy such as angioplasty or bypass surgery is indicated. This may be done during the initial hospitalization or later as an outpatient procedure based on the severity of results in the diagnostic tooling the MD orders (tests).

“SARS (severe acute respiratory syndrome, SARS-CoV-1) was a respiratory illness that spread in many countries around the world in 2002 and 2003. For many people, SARS caused flu-like symptoms (like fever and headache), but it progressed to severe illness in about 10% to 20% of people during the outbreak.

More than 8,000 people in 29 countries had SARS during the 2002-2003 outbreak. There was a small outbreak related to occupational exposure in 2004. Preventive measures stopped its spread, and only nine people were infected. There haven’t been any reported cases since then.

A virus called SARS-associated coronavirus (SARS-CoV-1) causes SARS.”

Cleveland Clinic (Severe Acute Respiratory Syndrome (SARS): Cause & Treatment)

  4 days ago

World Health Organization on this disease SARS coronavirus:

Severe acute respiratory syndrome (SARS) is a viral respiratory disease caused by a SARS-associated coronavirus. It was first identified at the end of February 2003 during an outbreak that emerged in China and spread to 4 other countries.

The virus identified in 2003. SARS-CoV is thought to be an animal virus from an as-yet-uncertain animal reservoir, perhaps bats, that spread to other animals (civet cats) and first infected humans in the Guangdong province of southern China in 2002.

Transmission

An epidemic of SARS affected 26 countries and resulted in more than 8000 cases in 2003. Since then, a small number of cases have occurred as a result of laboratory accidents or, possibly, through animal-to-human transmission (Guangdong, China).

Transmission of SARS-CoV is primarily from person to person. It appears to have occurred mainly during the second week of illness, which corresponds to the peak of virus excretion in respiratory secretions and stool, and when cases with severe disease start to deteriorate clinically. Most cases of human-to-human transmission occurred in the health care setting, in the absence of adequate infection control precautions. Implementation of appropriate infection control practices brought the global outbreak to an end.

Nature of the disease

Symptoms are influenza-like and include fever, malaise, myalgia, headache, diarrhoea, and shivering (rigors). No individual symptom or cluster of symptoms has proved to be specific for a diagnosis of SARS. Although fever is the most frequently reported symptom, it is sometimes absent on initial measurement, especially in elderly and immunosuppressed patients.

Cough (initially dry), shortness of breath, and diarrhoea are present in the first and/or second week of illness. Severe cases often evolve rapidly, progressing to respiratory distress and requiring intensive care.

Geographical distribution

The distribution is based on the 2002–2003 epidemic. The disease appeared in November 2002 in the Guangdong province of southern China. This area is considered as a potential zone of re-emergence of SARS-CoV.

Other countries/areas in which chains of human-to-human transmission occurred after early importation of cases were Toronto in Canada, Hong Kong Special Administrative Region of China, Chinese Taipei, Singapore, and Hanoi in Viet Nam.

Risk for travellers

Currently, no areas of the world are reporting transmission of SARS. Since the end of the global epidemic in July 2003, SARS has reappeared four times – three times from laboratory accidents (Singapore and Chinese Taipei), and once in southern China where the source of infection remains undetermined although there is circumstantial evidence of animal-to-human transmission.

Should SARS re-emerge in epidemic form, WHO will provide guidance on the risk of travel to affected areas. Travellers should stay informed about current travel recommendations. However, even during the height of the 2003 epidemic, the overall risk of SARS-CoV transmission to travellers was low.

Prophylaxis

None. Experimental vaccines are under development.

The National Institute of Health (NIH) states:

The virus that causes coronavirus disease 2019 (COVID-19) is stable for several hours to days in aerosols and on surfaces, according to a new study from National Institutes of Health, CDC, UCLA and Princeton University scientists in The New England Journal of Medicine. The scientists found that severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) was detectable in aerosols for up to three hours, up to four hours on copper, up to 24 hours on cardboard and up to two to three days on plastic and stainless steel. The results provide key information about the stability of SARS-CoV-2, which causes COVID-19 disease, and suggests that people may acquire the virus through the air and after touching contaminated objects. The study information was widely shared during the past two weeks after the researchers placed the contents on a preprint server to quickly share their data with colleagues.

The NIH scientists, from the National Institute of Allergy and Infectious Diseases’ Montana facility at Rocky Mountain Laboratories, compared how the environment affects SARS-CoV-2 and SARS-CoV-1, which causes SARS. SARS-CoV-1, like its successor now circulating across the globe, emerged from China and infected more than 8,000 people in 2002 and 2003. SARS-CoV-1 was eradicated by intensive contact tracing and case isolation measures and no cases have been detected since 2004. SARS-CoV-1 is the human coronavirus most closely related to SARS-CoV-2. In the stability study the two viruses behaved similarly, which unfortunately fails to explain why COVID-19 has become a much larger outbreak.

The NIH study attempted to mimic virus being deposited from an infected person onto everyday surfaces in a household or hospital setting, such as through coughing or touching objects. The scientists then investigated how long the virus remained infectious on these surfaces.

The scientists highlighted additional observations from their study:

• If the viability of the two coronaviruses is similar, why is SARS-CoV-2 resulting in more cases? Emerging evidence suggests that people infected with SARS-CoV-2 might be spreading virus without recognizing, or prior to recognizing, symptoms. This would make disease control measures that were effective against SARS-CoV-1 less effective against its successor.
• In contrast to SARS-CoV-1, most secondary cases of virus transmission of SARS-CoV-2 appear to be occurring in community settings rather than healthcare settings.  However, healthcare settings are also vulnerable to the introduction and spread of SARS-CoV-2, and the stability of SARS-CoV-2 in aerosols and on surfaces likely contributes to transmission of the virus in healthcare settings.

The findings affirm the guidance from public health professionals to use precautions similar to those for influenza and other respiratory viruses to prevent the spread of SARS-CoV-2:

• Avoid close contact with people who are sick.
• Avoid touching your eyes, nose, and mouth.
• Stay home when you are sick.
• Cover your cough or sneeze with a tissue, then throw the tissue in the trash.
• Clean and disinfect frequently touched objects and surfaces using a regular household cleaning spray or wipe.

The CDC states:

“-Seasonal influenza (flu) vaccines are designed to protect against the three influenza viruses that are expected to circulate most widely during the upcoming season.

-CDC is working with other federal agencies and the private sector, to improve flu vaccine technologies.

-Three different flu vaccine production technologies are FDA approved for use in the United States: egg-based flu vaccine, cell-culture based flu vaccine, and recombinant flu vaccine.

Influenza (flu) vaccines cause antibodies to develop in the body about two weeks after vaccination. These antibodies provide protection against infection with the influenza viruses that are used to make vaccine. The seasonal flu vaccine protects against the influenza viruses that research suggests will be most common during the upcoming season. These include three different main groups of influenza Type A and B viruses.”

Center for Disease Control and Prevention – CDC (How Influenza (Flu) Vaccines Are Made | Influenza (Flu) | CDC&Different Types of Flu Vaccines | Influenza (Flu) | CDC)

Influenza A (H1N1), Influenza A (H3N2), and one or two influenza B viruses (depending on the vaccine) are included in each year’s influenza vaccine now.

Learn how influenza got started:

 The 20th century alone 3 influenza pandemics occurred:

Spanish influenza in 1918 (~50+ million deaths),

Asian influenza in 1957 (two million deaths) and

Hong Kong influenza in 1968 (one million deaths).

FLU Pandemics – The 5 worst FLU outbreaks:

1- “The Russian Flu” -1889

Known as the “Russian Flu,” this influenza outbreak is believed to have begun in St. Petersburg but it soon spread across Europe and the world. It was one of the first epidemics that was covered regularly by the developing daily press. Newspapers wrote about the local spread of the disease and also discussed the situation in other distant European cities thanks to telegraph reports. It is estimated that around 1 million people died of the Russian Flu.

2-“The Spanish Flu” 1918-1919

Influenza was discovered not by a direct study of the disease in humans, but rather from studies on animal diseases. In 1918, J.S. Koen, a veterinarian, observed a disease in pigs which was believed to be the same disease as the now famous “Spanish” influenza pandemic of 1918.  If not the most severe pandemic than one of the most severe pandemics in history was the 1918 influenza virus, often called “the Spanish Flu.” The virus infected roughly 500 million people—one-third of the world’s population—and caused 50 million deaths worldwide (double the number of deaths in World War I). In the United States, a quarter of the population caught the virus, 675,000 died, and life expectancy dropped by 12 years. With no vaccine to protect against the virus, people were urged to isolate, quarantine, practice good personal hygiene, and limit social interaction.  The World Health Organization declared an outbreak of a new type of influenza A/H1N1 to be a pandemic in June 2009=Swine Flu.  Swine flu (H1N1) is a type of viral infection.  Swine flu it resembles a respiratory infection that pigs can get. Influenza—more specifically the Spanish flu—left its devastating mark in both world and American history that year. The microscopic killer circled the entire globe in four months, claiming the lives of more than 21 million people. The United States lost 675,000 people to the Spanish flu in 1918-more casualties possibly compared to World War I,  the Korean War and the Vietnam War combined not World War 2.   Pharmaceutical companies worked around the clock to come up with a vaccine to fight the Spanish flu, but they were too late. The virus disappeared before they could even isolate it.  It took 1/3 of the lives on earth.

Until February 2020, the 1918 epidemic was largely overlooked in the teaching of American history, despite the ample documentation at the National Archives and elsewhere of the disease and its devastation.

Over 100-years-old, from 1918, that just months ago seemed quaint and dated now seem oddly prescient. We make these records more widely available in hopes that they contain lessons about what to expect over the coming months and ideas about ways to avoid a repeat and prepare for what may follow.  H1N1-RX=VACCINE is the answer!

3-“Asian Flu”1957-1958

In February 1957, a new influenza A (H2N2) virus emerged in East Asia, triggering a pandemic (“Asian Flu”). This H2N2 virus was comprised of three different genes from an H2N2 virus that originated from an avian influenza A virus, including the H2 hemagglutinin and the N2 neuraminidase genes. It was first reported in Singapore in February 1957, Hong Kong in April 1957, and in coastal cities in the United States in summer 1957. The estimated number of deaths was 1.1 million worldwide and 116,000 in the United States.

Asian flu pandemic was a global pandemic of influenza A virus subtype H2N2 that originated in Guizhou in Southern China. The number of excess deaths caused by the pandemic is estimated to be 1–4 million around the world (1957–1958 and probably beyond), making it one of the deadliest pandemics in history.

4-“Hong Kong H3N2 Flu” 1968

The Hong Kong flu, also known as the 1968 flu pandemic, was a flu pandemic that occurred in 1968 and 1969-70 which killed between one and four million people globally.  It is among the deadliest pandemics in history, and was caused by an H3N2 strain of the influenza A virus. The virus was descended from H2N2 (which caused the Asian flu pandemic in 1957–1958) through antigenic shift, a genetic process in which genes from multiple subtypes are reassorted to form a new virus.  The first recorded instance of the outbreak appeared on 13 July 1968 in British Hong Kong. It has been speculated that the outbreak began in mainland China before it spread to Hong Kong;^[10] On 11 July, before the outbreak in the colony was first noted, the Hong Kong newspaper Ming Pao reported an outbreak of respiratory illness in Guangdong Province, and the next day, The Times issued a similar report of an epidemic in southeastern China.^[13] Later reporting suggested that the flu had spread from the central provinces of Sichuan, Gansu, Shaanxi, and Shanxi, which had experienced epidemics in the spring.  However, due to a lack of etiological information on the outbreak and a strained relationship between Chinese health authorities and those in other countries at the time, it cannot be ascertained whether the Hong Kong virus was to blame.  The outbreak lasted around six weeks, affecting about 15% of the population (some 500,000 people infected), but the mortality rate was low and the clinical symptoms were mild.

There were two waves of the flu in mainland China, one between July–September in 1968 and the other between June–December in 1970.  The reported data were very limited due to the Cultural Revolution, but retrospective analysis of flu activity between 1968 and 1992 shows that flu infection was the most serious in 1968, implying that most areas in China were affected at the time.

The epidemic became widespread in December, involving all 50 states before the end of the year.  Outbreaks occurred in colleges and hospitals, in some places the disease attacking upwards of 40% of their populations. Reports of absenteeism among students and nurses grew. Schools in Los Angeles, for example, reported rates ranging from 10 to 25%, compared to a typical 5 or 6%.  The Greater New York Hospital Association reported absenteeism of 15 to 20% among staff and urged its members to impose visitor restrictions to safeguard patients.  Economic activity was also hampered by high levels of industrial absenteeism.

Peak influenza activity for most states most likely occurred in the latter half of December or early January, but the exact week was impossible to determine due to the holiday season. Activity declined throughout January. Excess pneumonia-influenza mortality passed the epidemic threshold during the first week of December and increased rapidly over the next month, peaking in the first half of January. It took until late March for mortality to return to normal levels. There was no second wave during this season.  Following the epidemic of influenza A, outbreaks of influenza B began in late January and continued until late March. Mostly elementary-school children were affected.  This influenza B activity fit within the pattern of epidemics every three to six years, but the 1968–1969 flu season became the first documented instance of two major influenza A epidemics to occur in successive seasons.  Given the widespread epidemic levels of influenza A activity in 1968–1969, the CDC in June 1969 predicted little more than “sporadic cases” of influenza A in the 1969–1970 season.

The Hong Kong flu was the first known outbreak of the H3N2 strain, but there is serologic evidence of H3N1 infections in the late 19th century. The virus was isolated in Queen Mary Hospital located in Poc Fu Lam on Hong Kong Island of Hong Kong.

The estimates of the total death toll due to Hong Kong flu (from its beginning in July 1968 until the outbreak faded during the winter of 1969–70 vary:

• The World Health Organization and Encyclopaedia Britannica estimated the number of deaths due to Hong Kong flu to be between 1 and 4 million globally.
• The United States Centers for Disease Control and Prevention (CDC) estimated that, in total, the virus caused the deaths of 1 million people worldwide

However, the death rate from the Hong Kong flu was lower than most other 20th-century pandemics.

5-2009 H1N1 FLU

The 2009 swine flu pandemic, caused by the H1N1/swine flu/influenza virus and declared by the World Health Organization (WHO) from June 2009 to August 2010, was the third recent flu pandemic involving the H1N1 virus (the first being the 1918–1920 Spanish flu pandemic and the second being the 1977 Russian flu).   The first identified human case was in La Gloria, Mexico, a rural town in Veracruz. The virus appeared to be a new strain of H1N1 that resulted from a previous triple reassortment of bird, swine, and human flu viruses which further combined with a Eurasian pig flu virus,  leading to the term “swine flu” in this pandemic.

In 2009, an H1N1 pandemic infected millions of people worldwide.

Today, you can prevent H1N1 with an annual flu shot. You can treat it with rest, fluids and antiviral medications.  The 2009 H1N1 flu pandemic, also known as the swine flu, was the first major influenza outbreak of the 21st century.

Swine flu first appeared in Mexico and the United States in March and April 2009 and has swept the globe with unprecedented speed as a result of airline travel.

On June 11, 2009, the World Health Organization raised its pandemic level to the highest level, Phase 6, indicating widespread community transmission on at least two continents. The 2009 H1N1 virus contains a unique combination of gene segments from human, swine and avian influenza. This new H1N1 virus contained a unique combination of influenza genes not previously identified in animals or people. This virus was designated as influenza A (H1N1) virus. Ten years later work continued to better understand influenza, prevent disease, and prepare for the next pandemic.

Influenza may also affect other wild life which are horses, chickens and birds along with the pigs. In late 1917, military pathologists reported the onset of a new disease with high mortality that they later recognized as the flu. The overcrowded camp and hospital — which treated thousands of victims of chemical attacks and other casualties of war — was an ideal site for the spreading of a respiratory virus; 100,000 soldiers were in transit every day. It also was home to a live piggery, and poultry were regularly brought in for food supplies from surrounding villages. Oxford and his team postulated that a significant precursor virus, harbored in birds, mutated so it could migrate to pigs that were kept near the front.

Influenza A virus subtype H5N1, also known as A(H5N1) or simply H5N1, is a subtype of the influenza A virus which can cause illness in humans and many other animal species.  A bird-adapted strain of H5N1, called HPAI A(H5N1) for highly pathogenic avian influenza virus of type A of subtype H5N1, is the highly pathogenic causative agent of H5N1 flu, commonly known as avian influenza (“bird flu“).  It is enzootic (maintained in the population) in many bird populations, especially in Southeast Asia.

CDC Centers for Disease Control blog site states, “There are four types of influenza viruses: A, B, C and D. Human influenza A and B viruses cause seasonal epidemics of disease almost every winter in the United States. The emergence of a new and very different influenza A virus to infect people can cause an influenza pandemic. Influenza type C infections generally cause a mild respiratory illness and are not thought to cause epidemics. Influenza D viruses primarily affect cattle and are not known to infect or cause illness in people.

PMC U.S. National Library of Medicine (National Institutes of Health) states, “the announcement in 2005 that a virus causing fatal influenza during the great influenza pandemic of 1918–1919 had been sequenced in its entirety [1], in the laboratory of co-author JKT, has prompted renewed interest in the 1918 virus. The ongoing H5N1 avian influenza epizootic, and the possibility that it might also cause a pandemic [2], increase the importance of understanding what happened in 1918. However, in reviewing the scientific approach to unlocking an old puzzle, it is important to note that the sequencing of the 1918 virus took place after more than century of exhaustive and sometimes disheartening efforts to discover the cause of influenza (Figure 1). Indeed, the influenza search not only pre-dated the great pandemic of 1918, but also attracted the efforts of some of the greatest researchers of the 19th and 20th centuries. Along the way, the new fields of bacteriology and virology were advanced, and a productive marriage between microbiology, epidemiology and experimental science began. In describing here the 10-year effort (1995–2005) to sequence the genome of the 1918 pandemic influenza virus, we attempt also to place it within this important historical perspective.”

Influenza virus C is a genus in the virus family Orthomyxoviridae, which includes the viruses that cause influenza.  Nearly all adults have been infected with influenza C virus, which causes mild upper respiratory infections. Cold-like symptoms are associated with the virus including fever (38-40ᵒC=100.4 to 104F), dry cough, rhinorrhea (nasal discharge), headache, muscle pain, and achiness. The virus may lead to more severe infections such as bronchitis and pneumonia.  Lower tract complications are rare.  There is no vaccine against influenza C virus.

The species in this genus is called Influenza C virus. Influenza C viruses are known to infect humans and pigs.

Influenza D viruses primarily affect cattle and are not known to infect or cause illness in people.

THE CDC STATES:

• “The week ending January 25, 2024, was the first time that the percent of deaths for influenza (1.7%) was higher than the percent of deaths for COVID-19. The percent of deaths for influenza has continued to increase and is 2.6% for the week ending February 8, 2025.
• Eleven pediatric deaths associated with seasonal influenza virus infection were reported this week, bringing the 2024-2025 season total to 68 pediatric deaths.
• CDC estimates that there have been at least 29 million illnesses, 370,000 hospitalizations, and 16,000 deaths from flu so far this season.
• CDC recommends that everyone ages 6 months and older get an annual influenza (flu) vaccine.¹
• There are prescription flu antiviral drugs that can treat flu illness; those should be started as early as possible and are especially important for patients at higher risk for severe illness.²
• Influenza viruses are among several viruses contributing to respiratory disease activity. CDC is providing updated, integrated information about COVID-19, flu.”

Center for Disease Control and Prevention – CDC (Weekly US Influenza Surveillance Report: Key Updates for Week 6, ending February 8, 2025 | FluView | CDC)

As of January 2018 here is what was factual about the flu:

Influenza A still is the most common type of flu!

When someone dies directly from the flu, it happens very quickly, Adalja added.

The flu can also kill indirectly, meaning that the virus makes a person more susceptible to other health problems, and these health problems lead to death. For example, getting sick with the flu can make certain groups of people, such as older adults and people with chronic illnesses, more susceptible to bacteria that cause pneumonia, according to the Mayo Clinic. “Pneumonia is the most serious complication” of the flu and can be deadly, the Mayo Clinic says.

When a person with the flu gets pneumonia, the pneumonia is considered a secondary bacterial infection, Adalja said. (Pneumonia can be caused by either a virus or bacteria; in the case of a secondary infection after flu, it is caused by bacteria.) Death from such secondary infections usually occurs about a week or so after the person first got sick, because it takes time for the secondary infection to set in, Adalja said.

The flu can lead to death in other ways as well. People with the flu can experience “multiple organ failure” throughout their body (in order words, multiple organs stop working properly), which can be deadly, Adalja said.

The flu can also trigger other serious complications, including inflammation of the heart, brain or muscle tissues, according to the CDC. Infection can also lead to an extreme, body-wide inflammatory response known as sepsis, which can be life-threatening, the CDC says.

The 2017–2018 flu season has been particularly harsh, partially because the predominant strain of flu that’s spreading, H3N2, tends to cause more severe symptoms than other strains, Live Science reported the month of January 2018. And although the flu strains circulating this 2019 do match up with those covered in the season’s flu vaccine, an odd phenomenon may have occurred during the vaccine-making process inside chicken eggs. During that process, flu strains can acquire genetic changes, and this may have happened for the H3N2 component of the vaccine, Adalja said previously.

When we had not reached the end of February 2019 yet, we reached a point by CDC stating the following:

“Influenza activity in the United States during the 2017–2018 season began to increase in November and remained at high levels for several weeks during January–February. While influenza A(H3N2) viruses predominated through February, and were predominant overall for the season, influenza B viruses were more commonly reported starting in March, 2018. The season had high severity with unusually high levels of outpatient influenza-like illness, hospitalizations rates, and proportions of pneumonia and influenza-associated deaths.

CDC estimates that the burden of illness during the 2017–2018 season was also high with an estimated 48.8 million people getting sick with influenza, 22.7 million people going to a health care provider, 959,000 hospitalizations, and 79,400 deaths from influenza. The number of cases of influenza-associated illness that occurred last season was the highest since the 2009 H1N1 pandemic, when an estimated 60 million people were sick with influenza.

The 2017–2018 influenza season was additionally atypical in that it was severe for all ages of the population. The burden of influenza and the rates of influenza-associated hospitalization are generally higher for the very young and the very old, and while this was also true during the 2017–2018 season, rates of hospitalization in all age groups were the highest seasonal rates seen since hospital-based surveillance was expanded in 2005 to include all ages. This translated into an estimated 11.5 million cases of influenza in children, 30 million cases of influenza in working age adults (aged 18-64 years), and more than 7.3 million cases in adults aged 65 years and older.

Our estimates of hospitalizations and mortality associated with the 2017–2018 influenza season continue to demonstrate how severe influenza virus infection can be. We estimate overall, there were 959,000 hospitalizations and 79,400 deaths during the 2017–2018 season. More than 48,000 hospitalizations occurred in children (aged < 18 years); however, 70% of hospitalizations occurred in older adults aged ≥65 years. Older adults also accounted for 90% of deaths, highlighting that older adults are particularly vulnerable to severe disease with influenza virus infection. An estimated 10,300 deaths occurred among working age adults (aged 18–64 years), an age group that often has low influenza vaccination.”

CDC further states for 2024 and today the following:

• “The week ending January 25, 2024, was the first time that the percent of deaths for influenza (1.7%) was higher than the percent of deaths for COVID-19. The percent of deaths for influenza has continued to increase and is 2.6% for the week ending February 8, 2025.
• Eleven pediatric deaths associated with seasonal influenza virus infection were reported this week, bringing the 2024-2025 season total to 68 pediatric deaths.
• CDC estimates that there have been at least 29 million illnesses, 370,000 hospitalizations, and 16,000 deaths from flu so far this season.
• CDC recommends that everyone ages 6 months and older get an annual influenza (flu) vaccine.¹
• There are prescription flu antiviral drugs that can treat flu illness; those should be started as early as possible and are especially important for patients at higher risk for severe illness.²
• Influenza viruses are among several viruses contributing to respiratory disease activity. CDC is providing updated, integrated information about COVID-19, flu, and respiratory syncytial virus (RSV) activity on a weekly basis.”

What does this all mean?  Get the Influenza Vaccine for PREVENTION of the flu and Prevention of an Epidemic rising.  Help yourself and community!

References: CDC Centers for Disease Control and Prevention & Health Day