QUOTE FOR MONDAY:

“The word “pulmonary” means lung and the word “fibrosis” means scar tissue— similar to scars that you may have on your skin from an old injury or surgery. So, in its simplest sense, pulmonary fibrosis (PF) means scarring in the lungs. Over time, the scar tissue can destroy the normal lung and make it hard for oxygen to get into your blood.  Low oxygen levels (and the stiff scar tissue itself) can cause you to feel short of breath, particularly when walking and exercising. Pulmonary fibrosis isn’t just one disease. It is a family of more than 200 different lung diseases that all look very much alike.”

Pulmonary Fibrosis Foundation

 

QUOTE FOR THE WEEKEND:

“Your doctor will evaluate your signs and symptoms, review your medical history, and conduct a neurological examination. Signs and symptoms of a concussion may not appear until hours or days after the injury.  A cranial computerized tomography (CT) scan is the standard test to assess the brain right after a head injury. A CT scan uses a series of X-rays to obtain cross-sectional images of your skull and brain.”

MAYO CLINIC

Part II National Concussion Day-The symptoms/grade levels/diagnostic tooling with treatment and CTE=Chronic Traumatic Encephalopathy.

concussion 5   CTE2

Look at this done continuously  = This is a brain with CTE, above.

BEST CTE MUHAMMED  bEST CTE                   Mohammed Ali boxed in the head repeatedly over the years=CTE

CTE 4cte8 CTE 5 cte hockey

The signs and symptoms you may see indicating a concussion aren’t immediately present where in other situations observed immediately; it depends on the impact of the hit to the brain and location. Concussions are fairly common. Some estimates say a mild brain trauma is sustained every 21 seconds in the U.S. But it’s important to recognize the signs of a concussion so you can take the proper steps to treat the injury.

There are some common physical, mental, and emotional symptoms a person may display following a concussion. Any of these could be a sign of traumatic brain injury: confusion or feeling dazed/clumsiness/slurred speech/nausea or vomiting /headache /balance problems or dizziness /blurred vision/sensitivity to light/sensitivity to noise/sluggishness/Tinnitis – ringing in the ears/behavior or personality changes/concentration difficulties/memory loss.

Concussions are graded as mild (grade 1), moderate (grade 2), or severe (grade 3), depending on such factors as loss of consciousness, amnesia, and loss of equilibrium.

-Grade 1 concussion symptoms last for less than 15 minutes. There is no loss of consciousness.

-Grade 2 concussion there is no loss of consciousness but symptoms last longer than 15 minutes.

-Grade 3 concussion the person loses consciousness, sometimes just for a few seconds.

The seriousness of a concussion dictates what kind of treatment you should get. Most people with concussions fully recover with appropriate treatment. But because a concussion can be serious, safeguarding the person who got the concussion is important.  Seek medical attention. A health care professional can decide how serious the concussion is and whether you require treatment or not.

If you have suffered a grade 1 or grade 2 concussion, wait until symptoms are gone before returning to normal activities. That could take several minutes, hours, days, or even a week but still see a MD to know the level for sure.

If you have sustained a grade 1,2, or 3 concussion, see a doctor immediately for observation and treatment since most people don’t know the different levels or what level they have had. A doctor will be able to tell if all through diagnositic tooling. The M.D. will need to know details about how the concussion even happened including the symptoms you or the patient is having. The doctor asks simple common questions to evaluate memory and concentration skills (your name, the present year, where you are, etc…).

REGARDING DIAGNOSTIC TOOLING:

The doctor may test coordination and reflexes, which are both functions of the central nervous system. The doctor may also order a CT scan (computed tomography) or an MRI (magnetic resonance imaging) to rule out bleeding or other serious brain injury especially if he feels it was a grade 3 concussion.

If hospitalization is not required, the doctor will provide instructions for recovery. Aspirin-free medications may be prescribed and you will be advised to take it easy. Experts recommend follow-up medical attention within 24 to 72 hours if symptoms worsen.

  • Take a break. If your concussion was sustained during athletic activity, stop play and sit it out. Your brain needs time to properly heal, so rest is key. Definitely do not resume play the same day. Athletes and children should be closely monitored by coaches upon resuming play. If you resume play too soon, you risk a greater chance of having a second concussion, which can compound the damage. TheAmerican Academy of Neurology has issued guidelines about resuming activities after a concussion.
  • Guard against repeat concussions. Repeat concussions cause cumulative effects on the brain. Successive concussions can have devastating consequences, including brain swelling, permanent brain damage, long-term disabilities, or even death. Don’t return to normal activities if you still have symptoms. Get a doctor’s clearance so you can return to work or play with confidence. *                                                                                                                                      Can a concussion be prevented?*                                                                                                                                NO, since normally a concussion is unexpected, so it is tough to prevent. But there are several common-sense precautions you can take to lessen the possibility of traumatic brain injury. What you can do is PREVENTION by wearing proper protective equipment in high contact sports from football to hockey to boxing and even soccer (all increase the chance of a concussion). Bicycling, skateboarding, horseback riding to roller blading all can be a threat of a concussion due to a fall to the head. Wear head gear, padded guards for knees/elbows and even mouth gear or eye guards especially in racquet ball. Believe it or not a bike helmet can lower the risk of traumatic head injury by 85%. Ensure that the equipment is properly fitted, well maintained, and worn consistently.
  • For athletes and non athletes, always wear a seatbelt, obey posted speed limits, and don’t use drugs or alcohol, don’t be foolish texting or using the cell phone while driving because they can impair reaction time. Obviously, don’t fight to cause a blow to your head from occuring, and more males than females report traumatic head injuries.

Look what can happen after repeated blows done to the head over a long time to an athlete – Chronic traumatic encephalopathy (CTE) is a form of encephalopathy that is a progressive degenerative disease which can currently only be definitively diagnosed postmortem. Let us look at the meaning of the word actually in medical terminology=Chronic (meaning over a period of a long time) Traumatic (meaning the blows to the head) Encephalopathy (swelling of the brain definitely is a part of what happens but in all types of encephalopathy there is a brain malfunction. Know there are over 150 different terms that modify or precede “encephalopathy” in the medical literature. In March 2014, researchers announced the discovery of an exosome particle created by the brain which has been shown to contain trace proteins indicating the presence of the disease Baseline testing has been created to assess potential cognitive impairment in athletes in contact sports, but a test to determine the presence of CTE while the person is alive and a conventional postmortem diagnosis is not yet available. The disease was previously called dementia pugilistic, i.e. “punch-drunk”, as it was initially found in those with a history of boxing. CTE has been most commonly found in professional athletes participating in American football, Association football, ice hockey, professional wrestling and other contact sports who have experienced repetitive brain trauma. It has also been found in soldiers exposed to a blast or a concussive injury, in both cases resulting in characteristic degeneration of brain tissue and the accumulation of tau protein. Individuals with CTE may show symptoms of dementia, such as memory loss, aggression, confusion and depression, which generally appear years or many decades after the trauma.

CTE is a progressive degenerative disease of the brain found in people with a history of repetitive brain trauma, including symptomatic concussions as well as sub-concussive hits to the head that do not cause symptoms. In the case of blast injury, a single exposure to a blast and the subsequent violent movement of the head in the blast wind can cause the condition. Look at the perfect example of who this happened to that most of Americans know or heard about what the famous boxer Muhammed Ali.

Unfortunately chronic traumatic encephalopathy (CTE) at this time can only be diagnosed 100% after death. Below is a list of confirmed cases of CTE and the breakdown by sport. It should be noted that this is a small list compared to doctors understanding of the problem of CTE and the many undiagnosed cases. Through the work of the Center of the Study of Traumatic Encephalopathy researchers have been successful in securing donations of professional athletes brains after death for further study so researchers can better understand CTE and how to better prevent it. Many on this list were identified this way.

Professional Football Players:

  • John Grimsley – Houston Oilers, Miami Dolphins – February 2008 (45) – Case Study
  • Chris Henry – Cincinnati Bengals – December 2009
  • Dave Duerson – Chicago Bears – Suicide – February 17th, 2011 (50)
  • Lew Carpenter – Detroit Lions, Clevelend Browns, Green Bay Packers – November 14th, 2010 (78)
  • Lou Creekmur – Detroit Lions (NFL Hall of Fame) – July 5th, 2009 (82) – Case Study
  • Shane Dronett – Denver Broncos, Detroit Lions, Atlanta Falcons – January 12, 1971 Floyd Patterson

Professional Boxers:

Bobby Chacon/Jerry Quarry/Mike Quarry/Wilfred Benitez/Emile Griffith/Willie Pep/Sugar Ray Robinson/Billy Conn/Joe Frazier/Muhammad Ali (suspected)

If you notice in most of my articles there is a key to prevention in getting a illness or disease. Start taking the step towards prevention and not waiting till it happens especially CTE since the symptoms tell the M.D. the patient has high probability of the disease but can’t be actually 100% diagnosed till death in the morgue.

SO TAKE THE STEP IN MAKING A CONCUSSION NOT HAPPEN=PREVENTION, it is so simple.

QUOTE FOR FRIDAY:

“Concussions stand out as the single most common type of traumatic brain injury and is known by many names in the medical industry. None of them sound good, mild brain injury, mild traumatic brain injury, minor head trauma, not a thing any of us want to see listed on our medical report.”

Day of the year.com

Part I National Concussion Day-commonly overlooked; especially in sport. The NFL now even taking action.

concussion 4concussion 1

concussion 2concussion 3a

A concussion is a traumatic brain injury that alters the way your brain functions. Effects are usually temporary but can include headaches and problems with concentration, memory, balance and coordination.

Although concussions usually are caused by a blow to the head, they can also occur when the head and upper body are violently shaken. These injuries can cause a loss of consciousness, but most concussions do not. Because of this, some people have concussions and don’t realize it.

Concussions are common, particularly if you play a contact sport, such as football. But every concussion injures your brain to some extent. This injury needs time and rest to heal properly. Most concussive traumatic brain injuries are mild, and people usually recover fully.

******Remember the key to a brain concussion fully recovering is not to have impact to the head happening over and over again. Based on the same concept if you get hit in the same spot over and over again anywhere in the body bruising to actual injury will happen whether it be muscle or bone. Well get hit in the head over and over again like in sports especially boxing but now the big conversation is with football even with a helmet on you will cause a permanent damage to the brain.  A perfect example of this is a boxer that gets hit over an over again to the head in a boxing ring, take Muhammad Ali. The head is just another area of the body and no different than other areas of our body.******

What actually happens is this a concussion is most often caused by a sudden direct blow or bump to the head.

The brain is made of soft tissue. It’s cushioned by spinal fluid and encased in the protective shell of the skull. When you sustain a concussion, the impact can jolt your brain. Sometimes, it literally causes it to move around in your head. Traumatic brain injuries can cause bruising, damage to the blood vessels, and injury to the nerves.

The result? Your brain doesn’t function normally. If you’ve suffered a concussion, vision may be disturbed, you may lose equilibrium, or you may fall unconscious. In short, the brain is confused. That’s why Bugs Bunny often saw stars after getting whacked in the head in his cartoon by some other character.

The new uptake with football is being concerned with players getting concussions from getting hit by their opponent players whether it be defense or offense while playing the game. Concussions have become big business in the football world. With 1,700 players in the NFL, 66,000 in the college game, 1.1 million in high school and 250,000 more in Pop Warner, athletes and families across the country are eager to find ways to cut the risks of brain injury, whose terrifying consequences regularly tear across the sports pages. And a wave of companies offering diagnostic tools and concussion treatments are just as eager to sell them peace of mind.

That’s actually a slogan for one company. ImPACT, the maker of the world’s most popular concussion evaluation system, offers a 20-minute computerized test that players can take via software or online to measure verbal and visual memory, processing speed, reaction time and impulse control. The idea behind ImPACT (Immediate Post-Concussion Assessment and Cognitive Testing) and similar batteries is that doctors or athletic trainers can give a baseline test to a healthy athlete, conduct follow-up tests after an injury and then compare the results to help figure out when it’s OK to return the athlete to play. Selling itself as “Valid. Reliable. Safe,” ImPACT dominates the testing market and has spread throughout the sports world: Most NFL clubs use the test, as do all MLB, MLS and NHL clubs, the national associations for boxing, hockey and soccer in the U.S., and nine auto racing circuits.

A total of 87 out of 91 former NFL players have tested positive for the brain disease at the center of the debate over concussions in football, according to new figures from the nation’s largest brain bank focused on the study of traumatic head injury.

Researchers with the Department of Veterans Affairs and Boston University have now identified the degenerative disease known as chronic traumatic encephalopathy, or CTE, in 96 percent of NFL players that they’ve examined and in 79 percent of all football players. The disease is widely believed to stem from repetitive trauma to the head, and can lead to conditions such as memory loss, depression and dementia.

In total, the lab has found CTE in the brain tissue in 131 out of 165 individuals who, before their deaths, played football either professionally, semi-professionally, in college or in high school.

Forty percent of those who tested positive were the offensive and defensive linemen who come into contact with one another on every play of a game, according to numbers shared by the brain bank with FRONTLINE. That finding supports past research suggesting that it’s the repeat, more minor head trauma that occurs regularly in football that may pose the greatest risk to players, as opposed to just the sometimes violent collisions that cause concussions.

But the figures come with several important caveats, as testing for the disease can be an imperfect process. Brain scans have been used to identify signs of CTE in living players, but the disease can only be definitively identified posthumously. As such, many of the players who have donated their brains for testing suspected that they had the disease while still alive, leaving researchers with a skewed population to work with.

Even with those caveats, the latest numbers are “remarkably consistent” with past research from the center suggesting a link between football and long-term brain disease, said Dr. Ann McKee, the facility’s director and chief of neuropathology at the VA Boston Healthcare System.

“People think that we’re blowing this out of proportion, that this is a very rare disease and that we’re sensationalizing it,” said McKee, who runs the lab as part of a collaboration between the VA and BU. “My response is that where I sit, this is a very real disease. We have had no problem identifying it in hundreds of players.”

In a statement, a spokesman for the NFL said, “We are dedicated to making football safer and continue to take steps to protect players, including rule changes, advanced sideline technology, and expanded medical resources. We continue to make significant investments in independent research through our gifts to Boston University, the [National Institutes of Health] and other efforts to accelerate the science and understanding of these issues.”

The latest update from the brain bank, which in 2010 received a $1 million research grant from the NFL, comes at a time when the league is able to boast measurable progress in reducing head injuries. In its 2015 Health & Safety Report, the NFL said that concussions in regular season games fell 35 percent over the past two seasons, from 173 in 2012 to 112 last season. A separate analysis by FRONTLINE that factors in concussions reported by teams during the preseason and the playoffs shows a smaller decrease of 28 percent.

Tune in tomorrow for Part II Concussion-The symptoms/grade levels/diagnostic tooling with treatment and  more about CTE=Chronic Traumatic Encephalopathy.

QUOTE FOR THURSDAY:

“Atherosclerosis — often called hardening of the arteries — is the buildup of fatty substances in the arteries, which can lead to heart disease (One leading example is Heart Attack ) and stroke. It’s a slow, complex disease that typically starts in childhood and progresses with age.”

American Stroke Association

Get on it early with prevention, Childhood!

 

QUOTE FOR WEDNESDAY:

Prostate cancer affects more then 3 million men every year, worldwide. 1 in 9 men in the US diagnosed with prostate cancer in 2018.

Prostate Cancer Foundation

QUOTE FOR WEEKEND:

“The eye is like a tiny camera. Central Serous Retinopathy usually causes typical symptoms. It may be associated with either physical or emotional stress, and can occur at any age, although it is more commonly diagnosed in young adults (under 50 years old). Fluid can be detected in the macula with a dilated eye examination.”.

Retina Group

 

Central Serous Retinopathy

Central serous retinopathy (also called choroidopathy) is a relatively common condition in which a “blister” of fluid forms under the retina (serous retinal detachment). Since this “blister” forms in the center of the retina and affects central vision, the condition has been appropriately named central serous retinopathy (CSR).

The leakage is usually localized, but can be extensive. Individuals between the ages of 20 and 55 may develop this condition. Men are 10 times more likely than women to develop this condition. The typical symptoms may include blurred vision, distortion, altered color perception, reduced image size, and a central blind spot. The onset of symptoms is usually sudden. Vision may vary from 20/20 to 20/400.

The cause of CSR is unknown. Individuals with “hard-driving, type-A personalities” are more prone to develop this condition. On that basis, stress has been suggested as a possible cause, although this has never been proven.

Treatment

Treatment is usually not necessary because nearly 90% of individuals spontaneously recover 20/30 vision or better. The recovery usually occurs in one to six months. After recovery, some patients may still detect subtle visual deficits. For those patients in whom CSR does not resolve, laser eye surgery can be performed. Recurrences in the affected eye or the fellow eye are not uncommon.

The fluorescein angiogram test, or dye injection test, is invaluable in making the diagnosis, differentiating CSR from more serious problems, and in following atypical or prolonged cases.

QUOTE FOR FRIDAY:

“New Cases

  • Approximately every 3 minutes one person in the United States (US) is diagnosed with a blood cancer.
  • An estimated combined total of 176,200 people in the US are expected to be diagnosed with leukemia, lymphoma or myeloma in 2019.
  • New cases of leukemia, lymphoma and myeloma are expected to account for 10 percent of the estimated 1,762,450 new cancer cases diagnosed in the US in 2019.”

Leukemia and Lymphoma Society