“Epidermodysplasia verruciformis is usually an autosomal recessive inherited disorder.  Epidermodysplasia verruciformis is often due to mutations in the EVER1 or EVER2 genes on chromosome 17q25. The mutation decreases cell capacity to fight HPV infections.

The lesions usually start to appear during childhood and persist. About 7.5% of cases appear in infancy, 61.5% in children aged 5–11 years and 22.5% in puberty. The disease affects both males and females and people of all races.

Acquired epidermodysplasia verruciformis is a result of HIV infection, organ transplantation or a lymphoma.

Two clinical presentations: White, pink, reddish brown, dark brown or violet flat-topped papules and plaques with a scaly surface and irregular borders.  2-Verrucous or seborrhoeic keratoses-like lesions. These are raised brown warty lesions.”.

DernNet NZ all about skin (dernnetnz.org)